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Nicotinic acid has evolved from a pellagra-curing vitamin to a context‑dependent metabolic modulator, linking vitamin B₃ biology with NAD⁺ metabolism, lipid regulation, immunity, and host–microbiome interactions. Its history and mechanisms exemplify how nutritional discoveries have shaped modern precision approaches to cardiometabolic and inflammatory disease.
Read in this article about how exposomics and metabolomics together provide a powerful, complementary framework for advancing 5P medicine by linking environmental exposures with measurable metabolic phenotypes.
Eicosapentaenoic acid (EPA) is an omega‑3 polyunsaturated fatty acid that acts as a precursor to specialized pro‑resolving mediators, including resolvins, which actively dampen inflammatory responses.
Dimethylglycine is a molecular entry point into individualized, systems-oriented healthcare. Altered DMG levels have been linked to cardiometabolic risk and conditions such as myocardial infarction, diabetes, and metabolic dysfunction-associated liver disease.
Fructose is a simple sugar found in fruits and honey, but also added to many processed foods. Unlike glucose, it is mainly metabolized in the liver, and excessive intake can contribute to metabolic and inflammatory health issues.